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The symptoms are stimulated by the release of toxic bacterial components into the bloodstream when the bacteria break apart 3 4 5

However, the clinical efficacy of these treatments is often hampered by factors such as rapid drug clearance and inadequate drug delivery to the site of arterial injury 4,5,6

[1] The study found that treatment efficacy depended strongly on sex, drug type, time on treatment, and dose , with age having a weaker but measurable effect

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Indirect evidence for this possibility was provided by the high expression levels of the SA-induced gene PR1 in untreated and BSO+GSH-treated sym33-3 plants relative to other genotypes (Figure 7)