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These compounds facilitate the mobilization and removal of fat from the liver, preventing fat accumulation in this crucial organ

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Both peptides amplify glucose-stimulated insulin secretion, restrain glucagon, and are rapidly inactivated by the serine protease dipeptidyl-peptidase-4 (DPP-4)

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CSNK1a1 regulates PRMT1 to maintain the progenitor state in self-renewing somatic tissue