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010700), and UCHL1-eGFP (generated by the Ozdinler Laboratory at Northwestern Targeted Mutagenesis Core Facility, now also available at Jackson Laboratory, stock no

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Mechanistic studies have revealed that altered expression of iron metabolism-related proteins (including GPX4, FTH1, TfR1, and HO-1), accumulation of lipid peroxidation products, and disruption of antioxidant defense systems (particularly the Nrf2/GPX4 pathway) are central to ferroptosis progression in cardiac tissue

10.1179/1351000215Y.0000000015 Redox Rep

For personalised health advice, please consult a qualified healthcare professional.

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