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Invest Ophthalmol Vis Sci
For PTH1R 37 and GCGR 38 (whose crystal structures were determined with PGS domain fusion in ICL3 and T4L fusion at ICL2, respectively), agonist binding failed to promote the outward movement of the intracellular half of TM6 as seen in the inactive state, probably due to the fusion
The mechanism centers on TREK-1, a two-pore domain potassium (K2P) channel widely expressed in the central nervous system
Additionally, gene ontology analysis detected accumulation of transcripts related to the three steps of protein synthesis (initiation, elongation, termination) in Gclc fl/fl MZB compared to Gclc fl/fl FoB (Supplementary Fig
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Hayes MR, Skibicka KP, Grill HJ
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