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Ascorbic acid can compensate significantly for the adverse effects of aging in the ovary as was shown in mice [79], probably via inhibition of senescence through suppression of ROS production and AKT/mTOR signaling in mesenchymal stem cells [82]

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Interestingly, within the same study on lung cancer risk, the effect of the Pro/Pro genotype in conferring risk or protection differed according to smoking status, but only in those diagnosed with cancer at an older age (399)

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